Kidney damage in polycythemia Vera as a problem of interdisciplinary interaction


如何引用文章

全文:

开放存取 开放存取
受限制的访问 ##reader.subscriptionAccessGranted##
受限制的访问 订阅或者付费存取

详细

Polycythemia vera is a rare chronic progressive clonal myeloproliferative disorder. In polycythemia vera, the pathological process involves the bone marrow, spleen and liver; kidneys are extremely rare involved. In the case of kidney damage, various pathogenetic mechanisms are implemented, ranging from fairly obvious ones, due to the hyperuricemia characteristic of this disease, and ending with complex mechanisms with the development of glomerular damage. The article presents a clinical observation of a patient with polycythemia vera and kidney damage, the interpretation of which required the joint efforts of specialists in various fields.

全文:

受限制的访问

作者简介

Elena Voloshinova

Saratov State Medical University n.a. V.I. Razumovsky

Email: voloshinovaelena@mail.ru
Cand. Sci. (Med.), Associate Professor at the Department of Hospital Therapy, Faculty of General Medicine Saratov, Russia

Karina Safarova

Saratov State Medical University n.a. V.I. Razumovsky

Email: k.tsaturova@yandex.ru
Teaching Assistant at the Department of Hospital Therapy, Faculty of General Medicine Saratov, Russia

Elena Yakovleva

Saratov State Medical University n.a. V.I. Razumovsky

Email: elenaviktorova@yandex.ru
Cand. Sci. (Med.), Associate Professor at the Department of Hospital Therapy, Faculty of General Medicine Saratov, Russia

Svetlana Sazhnova

Saratov State Medical University n.a. V.I. Razumovsky

Email: sazhnova.svetlana@mail.ru
Cand. Sci. (Med.), Associate Professor at the Department of Hospital Therapy, Faculty of General Medicine Saratov, Russia

参考

  1. Гематология. Национальное руководство. Под ред. О.А. Рукавицына. М., 2017. С. 696- 718.
  2. McMullin M.F. Idiopathic erythrocytosis: a disappearing entity. Hematol. Am. Soc. Hematol. Educ. Program. 2009. Р. 629-635.
  3. Marchioli R., Finazzi G, Landolfi R., et al. Cardiovascular events and intensity of treatment in polycythemia vera. N. Engl. J. Med. 2013;368(1):22-33.
  4. Chen H., Zhang B., Li M., et al. Polycythemia vera associated with IgA nephropathy: A case report and literature review. Exp. Ther. Med. 2015;10:555-560. doi: 10.3892/etm.2015.2572.
  5. Aristizabal-Alzate A., Munoz-Grajales C., Ocampo-Kohn C., et al. Nefritis tubulointersticial y colangitis esclerosante asociadas a pancreatitis autoimmune. Nefrol. 2012;32:854-856. doi: 10.3265/Nefrologia.pre2012.Aug.11185.
  6. Ulusoy S., Ozkan G., Sönmez M., et al. Absence of hypoalbuminemia despite nephrotic proteinuria in focal segmental glomerulosclerosis secondary to polycythemia vera. Int. Med. 2010;49:2477-2480. Doi: 10.2169/ internalmedicine.49.4087.
  7. Kosch M., August C, Hausberg M., et al. Focal sclerosis with tip lesions secondary to polycythaemia vera. Nephrol. Dial. Transplant. 2000;15:1710-1711. doi: 10.1093/ndt/15.10.1710-a.

补充文件

附件文件
动作
1. JATS XML
下载
##common.cookie##