NUCLEOSIDE ANALOGUES IN THE TREATMENT OF DECOMPENSATED HEPATIC CIRRHOSIS CAUSED BY HEPATITIS B VIRUS


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Abstract

Chronic hepatic B virus (HBV) infection frequently leads to hepatic cirrhosis, whose decompensation reflects HBV reactivation under the influence of different exogenous and endogenous factors. The paper describes a case of reactivation of HBV infection in a female patient with chronic asymptomatc hepatic cirrhosis, rapidly developing liver cell failure and portal hypertension, as well as generalized vasculitis with the involvement of the aorta and its major branches (aortoarteritis) abolished by long-term entecavir treatment.

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About the authors

T. N LOPATKINA

I.M. Sechenov First Moscow State Medical University, Ministry of Health of Russia

Email: lopatkina-tn@mail.ru

E. L TANASHCHUK

E.M. Tareyev Clinic of Nephrology, Internal and Occupational Diseases, University Clinical Hospital Three

D. T ABDURAKHMANOV

I.M. Sechenov First Moscow State Medical University, Ministry of Health of Russia

T. A BAIKOVA

I.M. Sechenov First Moscow State Medical University, Ministry of Health of Russia

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