Email this article 
GIPOKSIYa KAK FAKTOR REGULYaTsII EKSPRESSII GENOV AROA-1, AVSA1 I S3 V MAKROFAGAKh ChELOVEKA
- Authors: Bogomolova AM1, Shavaa VS1, Nikitin AA1
-
Affiliations:
- Issue: Vol 16, No 4 (2016)
- Pages: 204-205
- Section: Articles
- URL: https://journals.eco-vector.com/MAJ/article/view/9716
- DOI: https://doi.org/10.17816/MAJ164204-205
- ID: 9716
Cite item
Open Access
Access granted
Abstract
Full Text
References
- Fong G.-H. Potential contributions of intimal and plaque hypoxia to atherosclerosis // CurrAtheroscler Rep.- 2015.- Vol. 17, № 6.- p. 510.
- Endogenous apolipoprotein A-I stabilizes ATP-binding cassette transporter A1 and modulates Toll-like receptor 4 signaling in human macrophages / Mogilenko D. A., Orlov S. V., Trulioff A. S., Ivanov A. V., Nagumanov V. K., Kudriavtsev I. V., Shavva V. S., Tanyanskiy D. A., Perevozchikov A. P. // FASEB j.- 2012.- Vol. 26, № 5.- P. 2019-2030.
- Complement in atherosclerosis: friend or foe? / Speidl W. S., Kastl S. P., Huber K., Wojta J. //J. Thromb. Haemost.- 2011.- Vol. 9, № 3.- P. 428-440.
- Inactivation of Tristetraprolin in Chronic Hypoxia Provokes the Expression of Cathepsin В / Fuhrmann D. C., Tausendschön M., Wittig I., Steger M., Ding M. G., Schmid T., Dehne N., Brüne В. // Mol Cell Biol.- 2015.- Vol. 35, № 3.- P. 619-630.
- Modified Low Density Lipoprotein Stimulates Complement C3 Expression and Secretion via Liver X Receptor and Toll-like Receptor 4 Activation in Human Macrophages / Mogilenko D. A., Kudriavtsev I. V., Trulioff A. S., Shavva V. S., Dizhe E. В., Missyul В. V., Zhakhov A. V., Ischenko A. M., Perevozchikov A. P., Orlov S. V. // J. Biol. Chem.- 2012.- Vol. 287, № 8.- P. 5954-5968.
- Retrovirus-mediated Expression of Apolipoprotein A-I in the Macrophage Protects against Atherosclerosis in Vivo / Ishiguro H., Yoshi da H., Major A. S., Zhu T., Babaev V. R., Linton M. F., Fazio S. //J. Biol. Chem.- 2001.- Vol. 276, № 39.- P. 36742-36748.
Supplementary files
СМИ зарегистрировано Федеральной службой по надзору в сфере связи, информационных технологий и массовых коммуникаций (Роскомнадзор).